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Terms in this set (153)

Elements of Effective High-Performance Team Dynamics

1. Clear Roles and Responsibilities
2. Knowing Your Limitations
3. Constructive Interventions
4. Knowledge Sharing
5. Summarizing and Reevaluating
6. Closed-Loop Communication
7. Clear Messages
8. Mutual Respect

Clear Roles and Responsibilities

Every member should know his/her role and responsibilities.

Resuscitation Triangle Roles

Compressor
AED/Monitor/Defibrillator
Airway

Leadership Roles

Team Leader
IV/IO Medications
Timer/Recorder

Knowing Your Limitations

Know your limitations, team leader should also know your limitations. Ask for advice when unsure.

Constructive Intervention

Leader may need to intervene if action may be inappropriate at the time. It should be tactful. Avoid confrontation.

Knowledge Sharing

Encourage an environment of knowledge sharing. Ask for good ideas for DDX. As if anything has been overlooked.

Summarizing and Reevaluating

Summarize information out load in a periodic update to the team. Review status of resuscitation attempt an announce plans for next steps

Closed-Loop Communications

Give clear message, orders, or assignments to team members.
Receive clear response from team members
Listen for conformation of response

Clear Message

concise communication spoke with distinctive speech in controlled tone of voice

Mutual Respect

Share a mutual respect for each other and work together

Systematic Approach

-If pt appears unconscious
- Use BLS Assessment for initial evaluation
- Then use Primary and Secondary assessments for more advanced evaluation and treatment

-If patient appears conscious
- Use the Primary Assessment for initial evaluation

BLS Assessment

Check Responsiveness

Shout for nearby help/activate Emergency response/AED/defibrillator

Check breathing and pulse

Defibrillation

Check Responsiveness

Tap and shout "Are you ok?"

Shout for nearby help/activate Emergency response/AED/defibrillator

Shout for nearby help
activate emergency response system
Get and AED if one is available, or send someone to get one

Check breathing and pulse

Check for absent or abnormal breathing by looking or scanning the chest for monument. for about 5-10 seconds

Pulse check should be performed simultaneously with breathing check to minimize delay in detection of cardiac arrest and initiation of CPR

Check pulse for 5-10 sec

If no pulse within 10 sec begin CPR with chest compressions

If there is a pulse start rescue breathing at 1 breath every 5-6 seconds. Check pulse every 2 minutes

Defibrillation

If no pulse, check for shockable rhythm

Provide shocks as indicated

Follow each show immediately with CPR beginning with compressions

How long do you try to limit interruptions in chest compressions

No longer than 10 seconds

Coronary Perfusion Pressure (CPP)

Aortic relaxation (diastolic) pressure - Right atrial relaxation (diastolic) pressure

ROSC

Return of Spontaneous Circulation

When is ROSC most likely to occur

When CPP of 15 mmHg or greater was achieved during CPR

Chest Compression Concepts

Compression of at least 2 inches

Compress the chest at rate of 100-120/min

Allow compete chest recoil after each compression

The Primary Assessment

Airway

Breathing

Circulation

Disability

Exposure

Airway

Maintain patent airway in unconscious patient using head tilt-chin lift, OPA or NPA

Used advanced airway management if needed

Breathing

Give O2 when indicated

Maintain O2 Sat of 94% or greater

Monitor adequacy of ventilation and oxygenation

Avoid excessive ventilation

Circulation

Monitor CPR quality
- Quantitative waveform capnography. if PETCO2 <10 mmHg, attempt to improve CPR quality

Attach monitor/defibrillator

Provide defibrillator/cardioversion

Obtain IV/IO

Give appropriate drugs

Give IV/IO fluids as needed

Check glucose and temp

Check perfusion issues

Disability

Check for neuro function

Quickly assess responsiveness, Level of consciousness, pupil dilation

AVPU: Alert, Voice, Painful, Unresponsive

Exposure

Remove clothing to perform a physical exam, looking for obvious sings of trauma, bleeding, burns, unusual markings, or medical alert brackets

Secondary Assessment

involves differential diagnosis, including focused history and search for underlying case.

SAMPLE: Signs/Symptoms, Allergies, Medications, PMH, Last meal consumed, Events

H's & T's

Potential reversible causes of cardiac arrest as well as emergency cardiopulmonary conditions

H's

Hypovolemia
Hypoxia
Hydrogen ion (acidosis)
Hypo-/hyperkalemia
Hypothermia

T's

Tension pneumothorax
Tamponade (cardiac)
Toxins
Thrombosis (pulmonary)
Thrombosis (coronary)

Most common causes of Pulseless Electrical Activity (PEA)

Hypovolemia and Hypoxemia

Hypovolemia

Common cause of PEA, initially produces a rapid narrow-complex tachycardia, and typically increases diastolic and decreased systolic pressure. As blood loss continues, blood pressure drops, eventually becoming undetectable, but narrow QRS complexes and rapid rate continue (PEA)

Respiratory Distress

Clinical state characterized by abnormal respiratory rate or effort

Respiratory Failure

Clinical state of inadequate oxygenation, ventral or both. Often the end stage of Respiratory Distress.

Respiratory Arrest

Cessation of breathing. Usually caused by events such as drowning or head injury .

Provide tidal volume approx 500-600 mL (6-7 mL/kg)

Excessive ventilation can cause

Gastric inflation

Regurgitation and aspiration

Increased intrathoracic pressure

Decreased venous return to heart

Diminished cardiac output and survival

Bag-Mask Ventilation

Deliver approximately 600 mL tidal volume to produce chest rise over 1 second

Oropharyngeal Airway (OPA)

Used in unconscious patient if chin lift or jaw thrust fails

Should NOT be used in conscious or semiconscious patient

How to select proper size OPA

Place OPA against side of face, when flange of OPA is at corner of moth, the tip should be at the angle of the mandible

Nasopharyngeal Airway (NPA)

Used as alternative to OPA

May be used in conscious, semiconscious or unconscious patients

How to select proper size NPA

Compare butter circumference of NPA with inner aperture of nare. Length of NPA should be same as distance from tip of patients nose to the earlobe.

Soft Suctioning

Aspiration of thin secretions fro oropharynx and nasopharynx

Performing intratracheal suctioning

Suctioning thought in-place airway (ie NPA) to access back of pharynx in pt with clenched teeth

Rigid Suctioning

More effective suction of the oropharynx, particularly if there is thick particulate matter (vomit)

Types of Advanced Airway devices

Laryngeal mask airway
Laryngeal tube
Esophageal-tracheal tube
ET tube

Ventilation rate of Advanced airway device

Cardiac Arrest: Once every 6 seconds

Respiratory Arrest: Once very 5-6 seconds

ACS Algorithm

STEMI Chain of Survival

Rapid recognition and reaction to STEMI warning signs

Rapid EMS dispatch and transport and prearrival notification to receiving hospital

Rapid assessment and dx in the ED (or cath lab)

Rapid treatment

Administer O2 and drugs for ACS/Suspected STEMI

Oxygen

Aspirin

Nitroglycerin

Opiates (e.g. Morphine)

Oxygen

give if O2<90%

Aspirin

-160-325 mg of non-enteric coated. Have patient chew them
-Give if pt has not taken ASA, allergy, or GI bleeding

Nitroglycerin

- 1 SL tablet or spray every 3-5 minutes.
-May repeat does twice (3 total doses)
- Administer only if hemodynamically stable
- SBP >90 mmHg or now lower than 30 mmHg below baseline
- HR is 50-100/min
Contraindications:
- Inferior wall MI and RV infarction
- Hypotension, bradycardia, or tachycardia
- Resecent phosphodiesterase inhibitor use

Morphine

Given for chest discomfort unresponsive to nitro

EKG

Obtain within 10 minutes of arrival and assess the patient

It is the center of decision pathway in management of ischemic chest discomfort and the only means to identify a STEMI

Reperfusion goals for STEMi patients

Fibrinolytic within 30 minutes of arrival or perform PCI within 90 minutes of arrival

The 1st 10 minutes of assessing and stabilizing patient

Check vitals
Establish IV
Brief focused H&P
Complete fibrinolytic checklist
Obtain labs
EKG
Portable Chest x-ray (less than 30 minutes after patients arrival in ED)

STEMI

ST-segment elevation in 2 or more contiguous leads or new LBBB.

J-joint elevation greater than 2mm in leads V2 and V3 and 1mm or more in all other leads by new or presumed new LBBB

2.5 mm in men younger than 40
1.5 mm in all women

NSTE-ACS

Ischemic ST-segment depression 0.5 mm or greater or dynamic T-wave inversion w/ pain or discomfort

Low-/intermediate-risk ACS

Normal or non diagnostic changes in T segment or T wave that are inconclusive and return future risk stratification

St-segment deviation in either direction of less that 0.5 mm or T-wave inversion less than or equal 2mm.

Mainstay tx for STEMI

Early reperfusion therapy achieved with primary PCI or fibrinolytic

Door to PCI Goal

90 minutes

if at non-PCI-capable hospital, 120 minutes

Door to Fibrinolytics

30 minutes

Fibrinolytic Agent

Clot buster, administered to pt with J-point ST segment elevation >2mm in leads V2 and V3, and 1 mm or more in all other leads or by new LBBB

examples: rTPA, reteplase, tenecteplase

Major types of Stroke

Ischemic Stroke- MC

Hemorrhagic stroke

Stroke Chain of survival

Rapid recognition and reaction to stroke warning signs

Rapid EMS dispatch

Rapid EMS system transport and prearrival notification

Rapid diagnosis and tx in hospital

8 D's of Stroke Care

1. Detection
2. Dispatch
3. Delivery
4. Door
5. Data
6. Decision
7. Drug/Devices
8. Disposition

NINDS in-hospital goals for assessment of suspect stroke

1. Immediate general assessment by the stroke team, emergency physical , or expert within 10 minutes of arrival, order surgery non contrast CT

2. Neurologic assessment by stroke team or designee and CT scan performed in 25 minutes of hospital arrival

3. Interpretation of the CT scan within 45 minutes of ED arrival

4. initial of fibrinolytic therapy in appropriate patients, within 1 hour of hospital arrival and 3 hours from symptom onset

5. Door to admission time of 3 hours

Cincinnati Prehospital Stroke Scale

Facial droop (have patient smile or try to show teeth)

Arm Drift (have patient close eyes and hold both arms out, with palms up)

Abnormal speech (have the patient say "you can't teach an old dog new tricks")

Immediate General Assessment and Stabilization

Assess ABCs

Provide Oxygen

Establish IV access and obtain blood samples

Check Glucose

Perform Neuro exam

Activate stroke team

Order CT scan

Obtain 12 lead EKG

Non contrast CT Scan

Can differentiate between ischemic and hemorrhagic stroke.

Most important test for a patient with acute stroke

If Hemorrhage is present

Pt not candidate for fibrinolytic

No Hemorrhage

Fibrinolytic, but if C/I give ASA

Fibrinolytic Therapy

Given to adults with acute ischemic stroke within 3 hours of onset of symptoms, or within 4.5 hours of onset of symptoms for selected patients

Inclusion Criteria for fibrinolytic

Dx of ischemic stroke causing neuro deficits

Onset of symptoms <3 hours

Age >18 years

Exclusion criteria for Fibrinolytic

Head trauma or stroke w/i 3 months
Symptoms of subarachnoid bleed
Arterial puncture in previous 7 days
History of previous intracranial hemorrhage
Elevated BP (>185 SBP and >110 DBP)
Active internal bleeding
Acute bleeding diathesis
Glucose <50
CT demonstrates multi lobar infarction

Inclusion Criteria for fibrinolytic 3-4.5 hr

ischemic stroke with neuro deficit
Onset of symptoms 3-4.5 hours before tx

Exclusion Criteria for Fibrinolytic 3-4.5 hr

Age >80
Severe Stroke (NIHSS score >25)
Taking oral anticoagulant regardless of INR
History of both DM and prior ischemic stroke

Monitor Blood Glucose

Consider giving IV or SQ insulin when glucose is greater than 185 mg/dL

Hypertension managent in rtPA candidates

Blood pressure must be 185 mmHg or less systolic and 110 mmHg or less Diastolic to limit risk of bleeding

Medications for treatment of HTN in stroke pt

Labetalol
Nicardipine

Labetalol dose

10-20 mg IV over 1-2 minutes, may repeat X 1

Nicardipine

IV 5mg/hr, titrate up to 2.5 mg/h every 15-20 minutes, max 15 mg/hr

Adult Cardiac Arrest Algorithm

Antiarrhythmics

Amiodarone
Lidocaine
Magnesium Sulfate

Amiodarone Dose

300 mg IV/IO bolus, then consider additional 150 mg IV/IO once

Class III Antiarrhythmic

Lidocaine Dose

Given if Amiodarone is unavailable

1-1.5 mg/kg IV/IO first dose, then 0.5 to 0.75 mg/kg IV/IO at 5-10 minute intervals

Max dose of 3 mg/kg

Magnesium Sulfate

1-2 g IV/IO diluted in 10 mL (D5W, NS) given as IV/IO bolus, typically over 5 to 20 minutes

Persistently low PETCO2 values less than 10mmHg

suggest ROSC is less likely to occur

Sudden spike in ETCO2 of 35-40 mmHG

Suggest ROSC has occurred

In Asystole consider stopping CPR if ETCO2 is

Less than 10 mmHg after 20 minute of CPR

Symptomatic Bradycardia

HR less than 50 w/ symptoms

Cornerstone of managing bradycardia

Differential beween S/S that are caused by the slow HR vs those that are unrelated

Correctly diagnose presence and type of AV blocks

Use Atropine as 1st line drug of choice

Decide when to intimate Transcutaneous pacing (TCP)

Decide when to start Eii or dopamine

Know when to cal expert consult

In addition: You must know the techniques and cautions for using TCP

Sinus Bradycardia w/ borderline first-degree AV block

Second-degree AV block type 1 (wenkebock)

Second-degree AV block type 2 (Mobitz)

Complete AV block w/ ventricular escape pacemaker

Third-Degree AV bock w/ junctional escape pacemaker

Adult Bradycardia With a Pulse Algorithm

Atropine

0.5 mg IV may repeat to a total dose of 3 mg

Dopamine Infusion

2 to 20 mcg/kg/min

Epinephrine Infusion

2 to 10 mcg/min

Avoid atropine in

Type II second-degree or third-degree AV blocks . These bradyarythmias are not likely to respond to reversal of cholinergic effects, tx is TCP or B-adrenergic support

Transcutaneous Pacing (TCP)

Delivers pacing impulses to heart throughout the skin by use of cutaneous electrodes.

When To use TCP

1. Hemodynamically unstable bradycardia
2. Unstable clinical condition like due to bradycardia
3. Pacing readiness in the setting of AMI as follows:
-Symptomatic sinus bradycardia
-Mobitz type II 2nd degree AV Block
-Third-degree AV block
- New Left, right, or alternator BBB, or bifacicular block
4. Bradycardia with symptomatic ventricular escape rhythms

Precaution for TCP

Contraindicated in severe hypothermia

not recommended for Asystole

Conscious patients require analgesia if time allows

Do not assess carotid pulse to confirm mechanical capture, electrical stimulation causes muscular jerking that may mimic the carotid pulse

Technique for TCP

1. Place pacing electrodes on chest
2. Turn the pacer on
3. Set the demand rate to approximately 60/min (can be adjusted up or down based on pt clinical response once pacing is established)
4. Set the current milliamperes output 2mA above the sole at which consistent capture is observed

Sinus tachycardia

Atrial fibrillation

Atrial flutter

Supraventricular tachycardia

Monomorphic ventricular tachycardia

Polymorphic ventricular tachycardia

2 Keys to management of tachycardia

1. Rapid recognition that the patient is significantly symptomatic or even unstable

2. Rapid recognition that the S/S are caused by the tachycardia

HR less than 150 is suggestive of

unlikely that symptoms of instability are caused by the tachycardia

HR greater than 150

usually inappropriate response to physiologic stress (fever, dehydration eat) or other underlying condition

Adult Tachycardia with a Pulse Algorithm

Unstable tachycardia

Requires immediate cardioversion, even in the absence of EKG

Most wide-complex tachycardias are...

Ventricular in origin

If pt unstable w/ pulse w/ regular uniform wide-complex vt

Treat with synchronized cardioversion and initial shock of 100J (monophasic waveform)

If no response to 1st shock, increase the dosage in a stepwise fashion

Arrhythmias w/ polymorphic QRS appearance (polymorphic VT)

Treat as VF w/ high-energy unsynchronized shock

Unsynchronized Shock

The electrical shock will be delivered as soon as operator pushes the shock button to discharge the device. These shocks use higher energy levels

Synchronized Shock

Uses a sensor to deliver a shock that is synchronized with a peak of the QRS complex

When to use Synchronized Shocks

Unstable VT

Unstable a-fib

Unstable a-flutter

Unstable regular monomorphic tachycardia w/ pulse

When to use unsynchronized shocks

Patient who is pulseless

Patient demonstrating clinical deterioration

When you are unsure whether monomorphic or polymorphic VT is present

Tx of choice when pt has symptomatic reentry SVT or VT with pulses

Synchronized cardioversion

Assessment of tachycardia w/ present pulse

Perform BLS assessment and Primary and Secondary assessments.

Determine if significant symptoms or sings are present and if they are due to the tachycardia

If S/S of tachycardia are due to the tachycardia

Pt is unstable and immediate cardioversion is indicated

If pt develops pulseless vt

deliver unsynchronized high-energy shocks and follow Cardiac arrest algorithm

Polymorphic VT

Treat the rhythm as VT and deliver high-energy unsynchronized shocks

Patient has stable tachycardia w/ pulse

BLS, Primary, Secondary assessment
- Look for sings of increased WOB and hypoxia
- Give O2; monitor O2 sat
- Support airway, breathing, circulation
-Obtain an EKG early to identify the rhythm; check BP
-Identify and treat reversible cause

Drugs to avoid in pt w/ Irregular Wide-complex tachycardia

AV nodal blocking agents such as Adenosine, CCB, Digoxin and possibly B-Blockers

Therapy for narrow QRS w/ regular rhythm is

Attempt Vagal maneuvers
Give adenosine

Vagal maneuvers

are preferred initial intervention for terminating narrow-complex tachycardia that are symptomatic (but stable) and supraventricular in origin

Adenosine

given if Vagal maneuvers fail

6mg rapid IV push over 1 second
followed by 20 mL saline flush

If SVT does not convert within 1-2 minutes
-Give second dose of adenosine 12 mg rapid IV push

Adenosine should generally not be given to

Asthma or COPD patients

Synchronized cardioversion for Narrow Regular

50-100 J

Synchronized cardioversion for Narrow Irregular

120-200 J

Synchronized cardioversion for Wide regular

100 J

Synchronized cardioversion for Wide irregular

Defibrillation dose NOT synchronized

Antiarrhythmic Infusions for Stable Wide-Complex Tachycardia

Procainamide
Amiodarone
Sotalol

Procainamide

20-50 mg/min until arrhythmia suppressed, hypotension ensures, QRS duration increased >50%, or maximum dose 17 mg/kg given

Maintenance infusion: 1-4 mg/min

Avoid of prolonged QT or CHF

Amiodarone (Infusion dose)

1st dose: 150 mg over 10 min
Repeat as needed if VT recurs

Follow by maintenance infusion of 1mg/min for first 6 hours

Sotalol

100 mg (1.5 mg/kg) over 5 min.

Avoid if prolonged QT

Drugs for post-cardiac arrest care

Epinephrine infusion
Dopamine infusion
Norepinephrine infusion

Adult Immediate Post-Cardiac Arrest Care Algorithm

Ventilation rate should be started at

10/min and titrated to achieve a PETCO2 of 35-40 mmHg or a PaCO2 of 40-45 mmHG

Targeted Temperature Management

Adults with ROSC after cardiac arrest, by selecting and maintaining a constant temperature between 32-36 degree C (89.6-95.2 degree F) for at least 24 hours

Treat Hypotension (SBP Less than 90 mmHg)

IV bolus

Norepinephrine

Epinephrine

Dopamine

IV Bolus for Hypotension

1-2 L normal saline or lactated Ringer's

Norepinephrine

0.1-0.5 mcg/kg/min IV infusion titrated to achieve minimum SBP >90 mmHg or MAP >65 mmHg

Epinephrine (for hypotension)

0.1-0.5 mcg/kg/min IV infusion titrated to achieve minimum SBP >90 mmHg or MAP >65 mmHg

Dopamine

5-10 mcg/kg/min IV infusion titrated to achieve minimum SBP >90 mmHg or MAP >65 mmHg

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Which is the best example of a role of the team leader?

Manage the work. The best leaders manage their team's work. They plan, organize, delegate, arrange resources, and ensure the completion of the team's responsibilities.

What is a step of closed loop communication ACLS?

By repeating back the verbal orders issued during a code, the loop of communication is closed. Closed-loop communication enables the person giving the orders to hear what they said reflected back and to confirm that their message was, in fact, received correctly.

What are the 3 signs of clinical deterioration that would cause activation of a rapid response system?

Each healthcare institutions establish their criteria when to activate the rapid response team but most of these criteria include: Heart rate less than 40 beats per minute. Heart rate greater than 130 beats per minute. A change in the systolic blood pressure to less than 90 mmHg.

What is mutual respect in ACLS?

Mutual Respect To have a high-performance team, everyone must abandon ego and respect each other during the resuscitation attempt, regardless of any additional training or experience that the team leader or specific team members may have.